mouse monoclonal anti human cyclin d1 Search Results


human/mouse cyclin d1/d2 antibody  (Bio-Techne corporation)
94
Bio-Techne corporation human/mouse cyclin d1/d2 antibody
Human/Mouse Cyclin D1/D2 Antibody, supplied by Bio-Techne corporation, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/human/mouse cyclin d1/d2 antibody/product/Bio-Techne corporation
Average 94 stars, based on 1 article reviews
human/mouse cyclin d1/d2 antibody - by Bioz Stars, 2026-02
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fitc-conjugated mouse anti-human cyclin b1  (Becton Dickinson)
90
Becton Dickinson fitc-conjugated mouse anti-human cyclin b1
Fitc Conjugated Mouse Anti Human Cyclin B1, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
fitc-conjugated mouse anti-human cyclin b1 - by Bioz Stars, 2026-02
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mouse anti human cyclin d1 monoclonal antibody  (MBL Life science)
90
MBL Life science mouse anti human cyclin d1 monoclonal antibody
Fenofibrate induces G1‐phase arrest through a blockade of the mitogen‐activated protein kinase (MAPK) cascade in HT‐29 cells. (a) Growth inhibitory effect of fenofibrate on HT‐29 cells. Cells were treated with fenofibrate at the indicated concentrations for 72 h, and viability was measured with a Cell Counting Kit‐8 assay. The data obtained with the solvent dimethyl sulfoxide (DMSO) was taken as 100%. Points, means (n = 3); bars, standard deviation (SD). **P < 0.01, compared with the DMSO‐treated control. (b) Cell cycle analysis of HT‐29 cells treated with fenofibrate. Cells were treated with fenofibrate at the indicated concentrations for 24 h. The DNA contents of the cells were analyzed by flow cytometry. The percentage in each phase of the cell cycle is shown. Columns, means (n = 3); bars, SD. **P < 0.01, compared with the DMSO‐treated control. (c) The effect of fenofibrate at 100 μΜ for 24 h on the phosphorylation status of ERK and the expression of <t>cyclin</t> <t>D1</t> analyzed by Western blotting. α‐tubulin was used as a loading control.
Mouse Anti Human Cyclin D1 Monoclonal Antibody, supplied by MBL Life science, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse anti human cyclin d1 monoclonal antibody/product/MBL Life science
Average 90 stars, based on 1 article reviews
mouse anti human cyclin d1 monoclonal antibody - by Bioz Stars, 2026-02
90/100 stars
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monoclonal mouse anti-human cyclin d1 protein antibody  (Becton Dickinson)
90
Becton Dickinson monoclonal mouse anti-human cyclin d1 protein antibody
Fenofibrate induces G1‐phase arrest through a blockade of the mitogen‐activated protein kinase (MAPK) cascade in HT‐29 cells. (a) Growth inhibitory effect of fenofibrate on HT‐29 cells. Cells were treated with fenofibrate at the indicated concentrations for 72 h, and viability was measured with a Cell Counting Kit‐8 assay. The data obtained with the solvent dimethyl sulfoxide (DMSO) was taken as 100%. Points, means (n = 3); bars, standard deviation (SD). **P < 0.01, compared with the DMSO‐treated control. (b) Cell cycle analysis of HT‐29 cells treated with fenofibrate. Cells were treated with fenofibrate at the indicated concentrations for 24 h. The DNA contents of the cells were analyzed by flow cytometry. The percentage in each phase of the cell cycle is shown. Columns, means (n = 3); bars, SD. **P < 0.01, compared with the DMSO‐treated control. (c) The effect of fenofibrate at 100 μΜ for 24 h on the phosphorylation status of ERK and the expression of <t>cyclin</t> <t>D1</t> analyzed by Western blotting. α‐tubulin was used as a loading control.
Monoclonal Mouse Anti Human Cyclin D1 Protein Antibody, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/monoclonal mouse anti-human cyclin d1 protein antibody/product/Becton Dickinson
Average 90 stars, based on 1 article reviews
monoclonal mouse anti-human cyclin d1 protein antibody - by Bioz Stars, 2026-02
90/100 stars
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mouse anti-human cyclin d1 monoclonal antibody  (Absolute Biotech Inc)
90
Absolute Biotech Inc mouse anti-human cyclin d1 monoclonal antibody
Fenofibrate induces G1‐phase arrest through a blockade of the mitogen‐activated protein kinase (MAPK) cascade in HT‐29 cells. (a) Growth inhibitory effect of fenofibrate on HT‐29 cells. Cells were treated with fenofibrate at the indicated concentrations for 72 h, and viability was measured with a Cell Counting Kit‐8 assay. The data obtained with the solvent dimethyl sulfoxide (DMSO) was taken as 100%. Points, means (n = 3); bars, standard deviation (SD). **P < 0.01, compared with the DMSO‐treated control. (b) Cell cycle analysis of HT‐29 cells treated with fenofibrate. Cells were treated with fenofibrate at the indicated concentrations for 24 h. The DNA contents of the cells were analyzed by flow cytometry. The percentage in each phase of the cell cycle is shown. Columns, means (n = 3); bars, SD. **P < 0.01, compared with the DMSO‐treated control. (c) The effect of fenofibrate at 100 μΜ for 24 h on the phosphorylation status of ERK and the expression of <t>cyclin</t> <t>D1</t> analyzed by Western blotting. α‐tubulin was used as a loading control.
Mouse Anti Human Cyclin D1 Monoclonal Antibody, supplied by Absolute Biotech Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse anti-human cyclin d1 monoclonal antibody/product/Absolute Biotech Inc
Average 90 stars, based on 1 article reviews
mouse anti-human cyclin d1 monoclonal antibody - by Bioz Stars, 2026-02
90/100 stars
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Fenofibrate induces G1‐phase arrest through a blockade of the mitogen‐activated protein kinase (MAPK) cascade in HT‐29 cells. (a) Growth inhibitory effect of fenofibrate on HT‐29 cells. Cells were treated with fenofibrate at the indicated concentrations for 72 h, and viability was measured with a Cell Counting Kit‐8 assay. The data obtained with the solvent dimethyl sulfoxide (DMSO) was taken as 100%. Points, means (n = 3); bars, standard deviation (SD). **P < 0.01, compared with the DMSO‐treated control. (b) Cell cycle analysis of HT‐29 cells treated with fenofibrate. Cells were treated with fenofibrate at the indicated concentrations for 24 h. The DNA contents of the cells were analyzed by flow cytometry. The percentage in each phase of the cell cycle is shown. Columns, means (n = 3); bars, SD. **P < 0.01, compared with the DMSO‐treated control. (c) The effect of fenofibrate at 100 μΜ for 24 h on the phosphorylation status of ERK and the expression of cyclin D1 analyzed by Western blotting. α‐tubulin was used as a loading control.

Journal: Cancer Science

Article Title: Novel MEK inhibitor trametinib and other retinoblastoma gene (RB)‐reactivating agents enhance efficacy of 5‐fluorouracil on human colon cancer cells

doi: 10.1111/cas.12139

Figure Lengend Snippet: Fenofibrate induces G1‐phase arrest through a blockade of the mitogen‐activated protein kinase (MAPK) cascade in HT‐29 cells. (a) Growth inhibitory effect of fenofibrate on HT‐29 cells. Cells were treated with fenofibrate at the indicated concentrations for 72 h, and viability was measured with a Cell Counting Kit‐8 assay. The data obtained with the solvent dimethyl sulfoxide (DMSO) was taken as 100%. Points, means (n = 3); bars, standard deviation (SD). **P < 0.01, compared with the DMSO‐treated control. (b) Cell cycle analysis of HT‐29 cells treated with fenofibrate. Cells were treated with fenofibrate at the indicated concentrations for 24 h. The DNA contents of the cells were analyzed by flow cytometry. The percentage in each phase of the cell cycle is shown. Columns, means (n = 3); bars, SD. **P < 0.01, compared with the DMSO‐treated control. (c) The effect of fenofibrate at 100 μΜ for 24 h on the phosphorylation status of ERK and the expression of cyclin D1 analyzed by Western blotting. α‐tubulin was used as a loading control.

Article Snippet: The following were used as the primary antibody: rabbit anti‐human p15 polyclonal antibody (C‐20, Santa Cruz Biotechnology, Santa Cruz, CA, USA), rabbit anti‐human p21 polyclonal antibody (C‐19, Santa Cruz Biotechnology), rabbit anti‐human p27 polyclonal antibody (C‐19, Santa Cruz Biotechnology), mouse anti‐human cyclin D1 monoclonal antibody (MBL, Nagoya, Japan), mouse anti‐human thymidylate synthase monoclonal antibody (Abcam, Cambridge, UK), rabbit anti‐human Akt, phospho‐Akt (Ser473), p42/44 MAPK, phospho‐p42/44 MAPK, phospho‐Rb (Ser780), and phospho‐Rb (Ser807/811) (Cell Signaling Technology).

Techniques: Cell Counting, Standard Deviation, Cell Cycle Assay, Flow Cytometry, Expressing, Western Blot